Student: Jeffrey
Medium: Sculpture
Jeffrey explains the creative side:
This chessboard represents a small portion of battle when a common form of bacteria attacks. The bacteria represent the white side as they commonly make the first move when enough bacteria cells have reproduced. The black side represents the cells, because your immune system can’t actually detect the bacteria until they change their movement patterns.
Jeffrey explains the scientific side:
The king of the immune system is the Dendritic Cell. They are one of the most important cells, because after taking a sample of the invaders, they decide whenever to release an army of anti-virus cells, or bacteria killers. The queen is the Helper T Cell. It activates the B-cells, killer T-cells, and the memory versions of both. They do the most, though they don’t contribute to the war effort directly. They help other cells, producing antibodies to work faster. They also prevent cells from overworking themselves, and activates other cells. Macrophages are the rooks of the immune system, because I’ve always described rooks as large, solid, and tough. Macrophages are huge compared to a normal cell, and they can usually take on about 100 bacteria cells. They are reliable and don’t offer drawbacks. The Neutrophils are the bishops of the immune sysytem. They arrive fast when help is wanted and cause a lot of damage. There is a large drawback as neutrophils commonly damage/ kill your body cells, so they have been designed to suicide after 5 days They are the second cells that come out to fight bacteria.The pawns are Antibodies. Antibodies tag invaders making them easy targets, or neutralize the invader its self. They are extremely small and plentiful, with B-cells making countless copies of them. They can be slightly modified to achieve different effects. The horses of the immune system are the monocytes. Monocytes are very versatile like the horse and travel quickly to sites of infection. They can change into macrophages if needed and can ease inflammation by removing dead cells from sites of infection.
